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SLC25A51 Decouples the Mitochondrial NAD+/NADH Ratio to control proliferation of AML cells. Lu et al.
Mar 1, 2024
Lu, Mu-Jie, 2024, "SLC25A51 Decouples the Mitochondrial NAD+/NADH Ratio to control proliferation of AML cells. Lu et al.", https://doi.org/10.18738/T8/DGLNSC, Texas Data Repository, V1, UNF:6:GgHj84WiC+iPfHOvqWZpKA== [fileUNF]
SLC25A51 selectively imports oxidized NAD+ into the mitochondrial matrix and is required for sustaining cell respiration. We observed an elevated expression of SLC25A51 that correlated with poorer outcomes in Acute Myeloid Leukemia (AML) patient data, and we sought to determine t...
Dynamics of SLC25A51 Reveal Preference for Oxidized NAD+ and Substrate Led Transport
Dynamics of SLC25A51 Reveal Preference for Oxidized NAD+ and Substrate Led Transport
Jun 16, 2023
Goyal, Shivansh, 2023, "Dynamics of SLC25A51 Reveal Preference for Oxidized NAD+ and Substrate Led Transport", https://doi.org/10.18738/T8/TIHMRO, Texas Data Repository, V2, UNF:6:fkBccDsQgMfL0Cu/AOs06A== [fileUNF]
SLC25A51 is a member of the mitochondrial carrier family (MCF) but lacks key residues that have been attributed contribute to the mechanism of other nucleotide MCF transporters. Thus, how SLC25A51 transports NAD+ across the inner mitochondrial membrane remains unclear. To elucida...
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