Bivalent chromatin domains in glioblastoma reveal a subtype-specific signature of glioma stem cells.
Hall AW, Battenhouse AM, Shivram H, Morris AR, Cowperthwaite MC, Shpak M, Iyer VR.
Cancer Res., 2018 March 16 [Epub ahead of print].
PMID: 29549165. doi: 10.1158/0008-5472.CAN-17-1724

Texas Data Repository DataSets include:

• Peak calls: MACS2 narrowPeak files for primary GBM tumor ChIP-seq experiments (doi:10.18738/T8/YJMLKO).
• Chromatin state: Outputs from ChromHMM chromatin state model, built from GBM tumor epigenetic and transcription factor marks (doi:10.18738/T8/UNDRTW).
• Gene expression: Tuxedo suite RNA-seq quantification files for GBM tumors and cell lines, produced by Cuffquant, Cuffnorm and Cuffdiff (doi:10.18738/T8/VVWHDG).
• Signal tracks: Normalized whole-genome ChIP-seq and RNA-seq bigWig signal tracks for hg38 (doi:10.18738/T8/QOWYPD, doi:10.18738/T8/BEXKWJ).

Original sequencing data deposited at dbGaP, Study phs001389.